Buffett was born in Omaha , Nebraska. He developed an interest in business and investing in his youth, eventually entering the Wharton School of the University of Pennsylvania in before transferring and graduating from the University of Nebraska at the age of He went on to graduate from Columbia Business School , where he molded his investment philosophy around the concept of value investing that was pioneered by Benjamin Graham. He attended New York Institute of Finance to focus his economics background and soon after began various business partnerships, including one with Graham. He created Buffett Partnership, Ltd in and his firm eventually acquired a textile manufacturing firm called Berkshire Hathaway , assuming its name to create a diversified holding company. In , Charlie Munger joined Buffett and became vice-chairman of the company. Buffett has been the chairman and largest shareholder of Berkshire Hathaway since Essentially, Buffett’s concentrated investments shelter managers from the short-term pressures of the market. He founded The Giving Pledge in with Bill Gates , whereby billionaires pledge to give away at least half of their fortunes.
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Many serious diseases have a genetic basis which, from an evolutionary point of view, should have been selected against, resulting in very low frequencies. The remarkable sustained prevalence of a number of disease-associated alleles is therefore surprising. We believe that antagonistic pleiotropy, when multiple effects of a gene have opposing effects on fitness e. We hypothesize that, rather than being an exception to the rule of genetic disorders, antagonistic pleiotropy may be common.
We surveyed the medical literature in order to determine whether sufficient evidence exists to reassess the nature of antagonistic pleiotropy; from being considered an unusual scenario to one that is anticipated. We also used a simple population genetic model to examine the feasibility of antagonistic pleiotropy to act as a mechanism to maintain polymorphism for serious genetic disorders even if the benefits are subtle.
We identified a number of examples of antagonistic pleiotropy where the deleterious effect, the beneficial effect, and the exact molecular cause have been demonstrated. We also identified putative cases in which there is circumstantial evidence or a strong reason to expect antagonistic pleiotropy in a genetic disorder. The population genetic model demonstrates that alleles with severe deleterious health effects can be maintained at medically relevant frequencies with only minor beneficial pleiotropic effects.
We believe that our identification of several cases of antagonistic pleiotropy, despite the lack of research on this question and the varied natures of the types of these disorders, speaks to both the underappreciated nature of this phenomenon and its potentially fundamental importance.
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To date, consideration of the implications of such antagonistically and recognition of, malaria resistance benefits due to the high profile of.
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